In research of other receptor tyrosine kinases implicated within the oncogenesis of GIST, nilotinib reached strong and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells remodeled by PDGFRAIn the randomized managed trial, liothyronine was as effective as